![Refining the amyloid β peptide and oligomer fingerprint ambiguities in Alzheimer's disease: Mass spectrometric molecular characterization in brain, cerebrospinal fluid, blood, and plasma - Michno - 2021 - Journal of Neurochemistry - Wiley Online Library Refining the amyloid β peptide and oligomer fingerprint ambiguities in Alzheimer's disease: Mass spectrometric molecular characterization in brain, cerebrospinal fluid, blood, and plasma - Michno - 2021 - Journal of Neurochemistry - Wiley Online Library](https://onlinelibrary.wiley.com/cms/asset/90191c73-6946-48c7-8f9a-7e6734a9aa10/jnc15466-fig-0002-m.jpg)
Refining the amyloid β peptide and oligomer fingerprint ambiguities in Alzheimer's disease: Mass spectrometric molecular characterization in brain, cerebrospinal fluid, blood, and plasma - Michno - 2021 - Journal of Neurochemistry - Wiley Online Library
![Mechanism of amyloid-β (Aβ) removal via Aβ-specific antibodies. FcR =... | Download Scientific Diagram Mechanism of amyloid-β (Aβ) removal via Aβ-specific antibodies. FcR =... | Download Scientific Diagram](https://www.researchgate.net/publication/330338281/figure/fig2/AS:720391836274692@1548766206812/Mechanism-of-amyloid-b-Ab-removal-via-Ab-specific-antibodies-FcR-Fc-receptors.jpg)
Mechanism of amyloid-β (Aβ) removal via Aβ-specific antibodies. FcR =... | Download Scientific Diagram
![Clinically accurate diagnosis of Alzheimer's disease via multiplexed sensing of core biomarkers in human plasma | Nature Communications Clinically accurate diagnosis of Alzheimer's disease via multiplexed sensing of core biomarkers in human plasma | Nature Communications](https://media.springernature.com/full/springer-static/image/art%3A10.1038%2Fs41467-019-13901-z/MediaObjects/41467_2019_13901_Fig1_HTML.png)
Clinically accurate diagnosis of Alzheimer's disease via multiplexed sensing of core biomarkers in human plasma | Nature Communications
![Biomedicines | Free Full-Text | Development of Alzheimer’s Disease Biomarkers: From CSF- to Blood-Based Biomarkers Biomedicines | Free Full-Text | Development of Alzheimer’s Disease Biomarkers: From CSF- to Blood-Based Biomarkers](https://www.mdpi.com/biomedicines/biomedicines-10-00850/article_deploy/html/images/biomedicines-10-00850-g001.png)
Biomedicines | Free Full-Text | Development of Alzheimer’s Disease Biomarkers: From CSF- to Blood-Based Biomarkers
![Amyloid β toxic conformer has dynamic localization in the human inferior parietal cortex in absence of amyloid plaques | Scientific Reports Amyloid β toxic conformer has dynamic localization in the human inferior parietal cortex in absence of amyloid plaques | Scientific Reports](https://media.springernature.com/full/springer-static/image/art%3A10.1038%2Fs41598-018-35004-3/MediaObjects/41598_2018_35004_Fig1_HTML.png)
Amyloid β toxic conformer has dynamic localization in the human inferior parietal cortex in absence of amyloid plaques | Scientific Reports
![Frontiers | Amyloid-β Peptide Impact on Synaptic Function and Neuroepigenetic Gene Control Reveal New Therapeutic Strategies for Alzheimer's Disease Frontiers | Amyloid-β Peptide Impact on Synaptic Function and Neuroepigenetic Gene Control Reveal New Therapeutic Strategies for Alzheimer's Disease](https://www.frontiersin.org/files/Articles/577622/fnmol-13-577622-HTML/image_m/fnmol-13-577622-g001.jpg)
Frontiers | Amyloid-β Peptide Impact on Synaptic Function and Neuroepigenetic Gene Control Reveal New Therapeutic Strategies for Alzheimer's Disease
![Amyloid Beta and Tau Cooperate to Cause Reversible Behavioral and Transcriptional Deficits in a Model of Alzheimer's Disease - ScienceDirect Amyloid Beta and Tau Cooperate to Cause Reversible Behavioral and Transcriptional Deficits in a Model of Alzheimer's Disease - ScienceDirect](https://ars.els-cdn.com/content/image/1-s2.0-S2211124719315268-fx1.jpg)
Amyloid Beta and Tau Cooperate to Cause Reversible Behavioral and Transcriptional Deficits in a Model of Alzheimer's Disease - ScienceDirect
![Plasma p-tau231 and p-tau217 as state markers of amyloid-β pathology in preclinical Alzheimer's disease | Nature Medicine Plasma p-tau231 and p-tau217 as state markers of amyloid-β pathology in preclinical Alzheimer's disease | Nature Medicine](https://media.springernature.com/full/springer-static/image/art%3A10.1038%2Fs41591-022-01925-w/MediaObjects/41591_2022_1925_Fig1_HTML.png)
Plasma p-tau231 and p-tau217 as state markers of amyloid-β pathology in preclinical Alzheimer's disease | Nature Medicine
Evaluating Amyloid-β Oligomers in Cerebrospinal Fluid as a Biomarker for Alzheimer's Disease | PLOS ONE
![Amyloid‐β oligomers in cellular models of Alzheimer's disease - Fontana - 2020 - Journal of Neurochemistry - Wiley Online Library Amyloid‐β oligomers in cellular models of Alzheimer's disease - Fontana - 2020 - Journal of Neurochemistry - Wiley Online Library](https://onlinelibrary.wiley.com/cms/asset/e5f275bd-b542-45d4-904f-bbfa43fbaf8d/jnc15030-fig-0004-m.jpg)
Amyloid‐β oligomers in cellular models of Alzheimer's disease - Fontana - 2020 - Journal of Neurochemistry - Wiley Online Library
![Cells | Free Full-Text | Circular RNA Encoded Amyloid Beta peptides—A Novel Putative Player in Alzheimer's Disease Cells | Free Full-Text | Circular RNA Encoded Amyloid Beta peptides—A Novel Putative Player in Alzheimer's Disease](https://pub.mdpi-res.com/cells/cells-09-02196/article_deploy/html/images/cells-09-02196-g001.png?1607045609)
Cells | Free Full-Text | Circular RNA Encoded Amyloid Beta peptides—A Novel Putative Player in Alzheimer's Disease
Secreted Amyloid β-Proteins in a Cell Culture Model Include N-Terminally Extended Peptides That Impair Synaptic Plasticity | Biochemistry
![Diagnostics | Free Full-Text | Nanobiosensors for Non-Amyloidbeta-Tau Biomarkers as Advanced Reporters of Alzheimer's Disease Diagnostics | Free Full-Text | Nanobiosensors for Non-Amyloidbeta-Tau Biomarkers as Advanced Reporters of Alzheimer's Disease](https://www.mdpi.com/diagnostics/diagnostics-10-00913/article_deploy/html/images/diagnostics-10-00913-g001.png)
Diagnostics | Free Full-Text | Nanobiosensors for Non-Amyloidbeta-Tau Biomarkers as Advanced Reporters of Alzheimer's Disease
![Amyloid-β misfolding as a plasma biomarker indicates risk for future clinical Alzheimer's disease in individuals with subjective cognitive decline | Alzheimer's Research & Therapy | Full Text Amyloid-β misfolding as a plasma biomarker indicates risk for future clinical Alzheimer's disease in individuals with subjective cognitive decline | Alzheimer's Research & Therapy | Full Text](https://media.springernature.com/lw685/springer-static/image/art%3A10.1186%2Fs13195-020-00738-8/MediaObjects/13195_2020_738_Fig1_HTML.png)
Amyloid-β misfolding as a plasma biomarker indicates risk for future clinical Alzheimer's disease in individuals with subjective cognitive decline | Alzheimer's Research & Therapy | Full Text
If amyloid drives Alzheimer disease, why have anti-amyloid therapies not yet slowed cognitive decline? | PLOS Biology
![Amyloid β deposition, neurodegeneration, and cognitive decline in sporadic Alzheimer's disease: a prospective cohort study - The Lancet Neurology Amyloid β deposition, neurodegeneration, and cognitive decline in sporadic Alzheimer's disease: a prospective cohort study - The Lancet Neurology](https://www.thelancet.com/cms/attachment/e4e3b80c-a0d0-4767-9e98-452f98002700/gr1_lrg.jpg)
Amyloid β deposition, neurodegeneration, and cognitive decline in sporadic Alzheimer's disease: a prospective cohort study - The Lancet Neurology
![Amyloid-β42/40 ratio drives tau pathology in 3D human neural cell culture models of Alzheimer's disease | Nature Communications Amyloid-β42/40 ratio drives tau pathology in 3D human neural cell culture models of Alzheimer's disease | Nature Communications](https://media.springernature.com/full/springer-static/image/art%3A10.1038%2Fs41467-020-15120-3/MediaObjects/41467_2020_15120_Fig1_HTML.png)